Download PDF by Lee L. Swanstrom, Christy M. Dunst: Antireflux Surgery

By Lee L. Swanstrom, Christy M. Dunst

This quantity presents a finished, state of the art evaluation of the main concerns particular to the sector of antireflux surgical procedure. It presents unprecedented tutorial element concerning functionality of antireflux surgical procedure from major esophageal surgeons worldwide. This quantity represents the single source of its style committed in particular to the problems exact to antireflux surgical procedure. it really is wealthy intimately and worthwhile illustrations that coach surgeons in right process in addition to providing the reasoning at the back of a number of ideas. Written via specialists within the box, Antireflux surgical procedure is of significant price to working towards surgeons who practice gastrointestinal surgical procedure, clinical scholars, surgical citizens, and fellows.

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Hum Pathol. 1992;23:475–6. 42. Pohl H, Welch HG. The role of overdiagnosis and reclassification in the marked increase of esophageal adenocarcinoma incidence. J Natl Cancer Inst. 2005;97:142–6. 43. Chandrasoma PT, Wickramasinghe K, Ma Y, DeMeester TR. Adenocarcinomas of the distal esophagus and “gastric cardia” are predominantly esophageal adenocarcinomas. Am J Surg Pathol. 2007;31:569–75. 44. Rice TW, Blackstone EW, Rusch VW. 7th edition of the AJCC cancer staging manual: esophagus and esophagogastric junction.

Because the criteria for their definition are simple (based on the presence or absence of three easily recognizable cell type: mucous cells, parietal cells, and goblet cells), their 2 Effects of Reflux on the Esophageal Mucosa identification in biopsies is easy, precise, and accurate with little inter-observer variation after minimal training. Together, various combinations of these three columnar epithelial types comprise the entire pathologic metaplastic gap that results from columnar metaplasia of squamous epithelium [1, 40].

If intestinal metaplasia is present, it is almost always present in the most proximal region of the gap immediately adjacent to the squamo-columnar junction. When present, the amount of intestinal metaplasia varies greatly in different patients. In some patients intestinal metaplasia is limited to the most proximal region of the gap; in others, the intestinal metaplasia extends distally to involve an increasing part of the gap. The involvement is usually contiguous without skip areas. In a few patients, intestinal metaplasia is present in the entire gap but there is usually non-intestinalized cardiac and oxyntocardiac mucosa in the most distal part of the gap separating intestinal from gastric oxyntic mucosa.

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