Read e-book online Colorectal Cancer (2006) PDF

By Jim Cassidy, Patrick Johnston, Eric Van Cutsem

Written by means of international experts at the topic, this reference comprehensively covers each point of colorectal melanoma. Addressing the molecular mechanisms, genetics, identity, and treatment of ailment, this advisor covers new pharmaceutical advancements, present screening protocols, and sleek equipment of illness administration through experts at well known associations equivalent to the Royal Marsden health facility, the Memorial Sloan Kettering melanoma heart, and the Vanderbilt-Ingram melanoma middle.

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57. Aarnio M, Mustonen H, Mecklin JP, Jarvinen HJ. Prognosis of colorectal cancer varies in different high-risk conditions. Ann Med 1998; 30:75–80. 58. Sankila R, Aaltonen LA, Jarvinen HJ, Mecklin JP. Better survival rates in patients with MLH1-associated hereditary colorectal cancer. Gastroenterology 1996; 110:682–687. 59. Watson P, Lin KM, Rodriguez-Bigas MA, et al. Colorectal carcinoma survival among hereditary nonpolyposis colorectal carcinoma family members. Cancer 1998; 83:259–266. 60. Lin KM, Shashidharan M, Ternent CA, et al.

Nagase H, Nakamura Y. Mutations of the APC (adenomatous polyposis coli) gene. Hum Mutat 1993; 2:425–434. 99. Mandl M, Paffenholz R, Friedl W, Caspari R, Sengteller M, Propping P. Frequency of common and novel inactivating APC mutations in 202 families with familial adenomatous polyposis. Hum Mol Genet 1994; 3:181–184. 100. Smith KJ, Johnson KA, Bryan TM, et al. The APC gene product in normal and tumor cells. Proc Natl Acad Sci USA 1993; 90:2846–2850. 101. Nathke IS, Adams CL, Polakis P, Sellin JH, Nelson WJ.

Loss of imprinting of the insulin-like growth factor II gene (IGF2) has been found in the normal colonic mucosa in around one-third of patients with colorectal cancer, a threefold excess in comparison to controls (164). 7) in circulating lymphocytes, indicating it may be a marker for colorectal cancer. However, further evidence also indicates that not only is loss of IGF2 imprinting heritable but it also is associated with increased tumor burden in an animal model (165). Thus it is possible that epigenetic mechanisms are also involved in colorectal cancer susceptibility, further complicating attempts to identify the genes contributing to the disease burden.

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